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Peripheral neurological lesions in rheumatoid arthritis were first reported by Pitres and Vaillard in 1887 in France and later by Bannatyne in 1898 (Hart and Golding 1960). Subsequently only sporadic references have appeared. In a sufferer of rheumatoid arthritis with painful, stiff and swollen joints, the additional peripheral neurological features may make life more unbearable. Sometimes the neurological symptoms become the patientâ€™s chief concern and neuropathy may itself be the cause of locomotor disability. Although rheumatoid arthritis is much commoner in women, rheumatoid neuropathy is at least as common in men (Hart and Golding 1960, Pallis and Scott 1965, Chamberlain and Bruckner 1970). Defective peripheral circulation and disturbed blood supply to the affected joints are apparent in many cases of chronic arthritis. The disturbance of peripheral circulation in these cases involves chiefly the minute vessels and may lead to trophic changes in the extremities (Kovaks 1934, Yadav 1975). Symptoms due to ischaemia may be recorded either because of prolonged spasm or small but multiple occlusive lesions of the digital arteries (Hart and Golding 1960, Laws et al. 1967, Yadav 1976). The nail edge and other soft tissue lesions commonly observed in rheumatoid arthritis are also associated with peripheral neuropathy. These changes have been found to be due to obliterative endarteritis (By Waters 1964). In this study arteriographic findings in 18 known cases of rheumatoid arthritis with various types of peripheral neuropathy have been described.